According to research, a common cold may provide some protection from Covid.
According to study, natural defenses against a common cold may also offer some protection against Covid-19.
According to study, natural defenses against a common cold may also offer some protection against Covid-19.
The small-scale study, which was published in Nature Communications, enlisted the help of 52 people who resided with someone who had recently contracted Covid-19.
After a cold, those who formed a “memory bank” of certain immune cells to help avoid future attacks looked to be less likely to develop Covid.
Experts believe that no one should rely only on this defense, and that immunizations will continue to be important.
They believe, however, that their findings may provide significant insight into how the body’s immune system combats the infection.
Because Covid-19 is caused by a type of coronavirus, and other colds are caused by other coronaviruses, scientists have questioned if immunization to one would help prevent the spread of another.
However, doctors warn that thinking that anyone who has recently had a cold is automatically protected against Covid-19 is a “fatal mistake,” as not all colds are caused by coronaviruses.
The Imperial College London researchers sought to know why some people get Covid after being exposed to it and others don’t.
‘A new vaccine strategy’
They concentrated their research on T-cells, which are an important aspect of the immune system. Some of these T-cells are capable of killing any cells infected with a specific threat, such as a cold virus.
After the cold has passed, some T-cells remain in the body as a memory bank, ready to develop a defense when the virus reappears.
Researchers evaluated 52 people who had not yet been vaccinated but lived with people who had just tested positive for Covid-19 in September 2020.
During the 28-day study period, half of the group received Covid and the other half did not.
A third of those who were not infected with Covid had significant amounts of particular memory T-cells in their blood.
These were most likely formed when the body was infected with another closely related human coronavirus – most commonly, a common cold, according to the researchers.
Other factors, like as ventilation and how contagious their household contact was, could also influence whether someone contracted the illness, according to the researchers.
Although this was a tiny study, Dr Simon Clarke of the University of Reading said it adds to our understanding of how our immune system fights the virus and could help with future vaccinations.
“These facts should not be over-interpreted,” he added. It’s doubtful that someone who has died or has had a more serious infection has never experienced a coronavirus-caused cold.
“It could also be a major mistake to believe that everybody who has recently had a cold is immune to Covid-19, because coronaviruses account for only 10-15 percent of colds.”
Professor Ajit Lalvani, the study’s senior author, concurred that immunizations were essential for protection.
“Learning from what the body does well could help inform the design of new products,” he added.
Current vaccines target spike proteins on the virus’s outer surface, but these spike proteins can change when new varieties emerge.
T-cells in the body, on the other hand, target internal virus proteins, which do not vary as much from one version to the next, implying that vaccinations that better harness the action of T-cells could provide broader, longer-lasting protection against Covid, he added.